Skeletal Muscle

雜志介紹

Skeletal Muscle雜志介紹

《Skeletal Muscle》是一本以English為主的開放獲取國際優秀期刊，中文名稱骨骼肌，本刊主要出版、報道醫學-CELL BIOLOGY領域的研究動態以及在該領域取得的各方面的經驗和科研成果，介紹該領域有關本專業的最新進展，探討行業發展的思路和方法，以促進學術信息交流，提高行業發展。該刊已被國際權威數據庫SCIE收錄，為該領域相關學科的發展起到了良好的推動作用，也得到了本專業人員的廣泛認可。該刊最新影響因子為5.3，最新CiteScore 指數為9.1。

本刊近期中國學者發表的論文主要有：

• IMB0901 inhibits muscle atrophy induced by cancer cachexia through MSTN signaling pathway

  Author: Dong Liu, Xinran Qiao, Zhijuan Ge, Yue Shang, Yi Li, Wendie Wang, Minghua Chen, Shuyi Si, Shu-zhen Chen

• Metabolic derangements of skeletal muscle from a murine model of glioma cachexia

  Author: Pengfei Cui, Wei Shao, Caihua Huang, Chang-Jer Wu, Bin Jiang, Donghai Lin

• Retinoid acid-induced <Emphasis Type="Italic">microRNA-31-5p</Emphasis> suppresses myogenic proliferation and differentiation by targeting <Emphasis Type="Italic">CamkIIδ</Emphasis>

  Author: Bo Liu, Chao Liu, Wei Cong, Nan Li, Nan Zhou, Yi Tang, Chao Wei, Han Bai, Ying Zhang, Jing Xiao

• Low-intensity pulsed ultrasound prevents muscle atrophy induced by type 1 diabetes in rats

  Author: Liang Tang, Nan Li, Wenqi Jian, Yiting Kang, Bo Yin, Shuxin Sun, Jianzhong Guo, Lijun Sun, Dean Ta

英文介紹

Skeletal Muscle雜志英文介紹

The only open access journal in its field, Skeletal Muscle publishes novel, cutting-edge research and technological advancements that investigate the molecular mechanisms underlying the biology of skeletal muscle. Reflecting the breadth of research in this area, the journal welcomes manuscripts about the development, metabolism, the regulation of mass and function, aging, degeneration, dystrophy and regeneration of skeletal muscle, with an emphasis on understanding adult skeletal muscle, its maintenance, and its interactions with non-muscle cell types and regulatory modulators.

Main areas of interest include:

-differentiation of skeletal muscle-

atrophy and hypertrophy of skeletal muscle-

aging of skeletal muscle-

regeneration and degeneration of skeletal muscle-

biology of satellite and satellite-like cells-

dystrophic degeneration of skeletal muscle-

energy and glucose homeostasis in skeletal muscle-

non-dystrophic genetic diseases of skeletal muscle, such as Spinal Muscular Atrophy and myopathies-

maintenance of neuromuscular junctions-

roles of ryanodine receptors and calcium signaling in skeletal muscle-

roles of nuclear receptors in skeletal muscle-

roles of GPCRs and GPCR signaling in skeletal muscle-

other relevant aspects of skeletal muscle biology.

In addition, articles on translational clinical studies that address molecular and cellular mechanisms of skeletal muscle will be published. Case reports are also encouraged for submission.

Skeletal Muscle reflects the breadth of research on skeletal muscle and bridges gaps between diverse areas of science for example cardiac cell biology and neurobiology, which share common features with respect to cell differentiation, excitatory membranes, cell-cell communication, and maintenance. Suitable articles are model and mechanism-driven, and apply statistical principles where appropriate; purely descriptive studies are of lesser interest.